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How Strong Is ACP105? To get a clear representation of just how strong of an effect this SARM has, each research subject undergoes ORX-induced atrophy (an orchiectomy), which is a surgical procedure in which one or both testicles are removed. This is the surgical form of castration. The point of this is that once the ACP105 is administered to the research subjects, you can calculate an exact figure that defines how anabolic and androgenic a compound is, based on the fact that there are no longer endogenous hormones skewing the results, and the research subject is essentially functioning from a baseline reading of 0 on all accounts. 1mg/kg per day was given to the research subjects, and that resulted in a very blatant anabolic effect. The extent of this anabolic effect is measured in what % of hypertrophy occurs relative to the baseline that DHT would otherwise achieve had the research subject not undergone ORX-induced atrophy. The % of anabolic activity was determined to be 67%, with only 21% reversal of the prostate gland (which is used to measure androgenic activity). From this we can conclude that this SARM has an anabolic:androgenic rating/ratio of 3.19:1 There are several SARMs that have anabolic:androgenic ratios of 3:1 and up though, so this is fairly typical. While we can vaguely gather what we can expect from this compound (the comparisons that come to mind are Ostarine and S4), without knowing the binding affinity of the compound we can essentially only make shot in the dark guesses. We do know what kind of interaction ACP105 has with the androgen receptor though, which helps shed light on things a bit more. Based on the information that was available, we know that ACP105 is a partial agonist. Full agonists induce greater levels of suppression, but are more potent (based on the information presented). Partial agonists on the other hand, are less suppressive, but are typically weaker relative to Testosterone and DHT, which is the case with AC
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